Monogamous prairie voles have far more oxytocin receptors in the nucleus accumbens than non-monogamous voles, which explains why they work harder to reunite with a separated mate.
Snapshot · Huberman Lab
Monogamous prairie voles have far more oxytocin receptors in the nucleus accumbens than non-monogamous voles, which explains why they work harder to reunite with a separated mate.
Where this was said
At 21:30 · chapter starts 18:21
Prairie voles are an unlikely star of grief neuroscience, but Huberman makes the case compellingly. Within the same species, some populations are monogamous — bonding for life, raising young together — while others are non-monogamous. When experimenters separate a bonded pair with a physical barrier, the monogamous voles will work intensely to breach it and return to their partner; the non-monogamous voles simply do not. [1] — Andrew Huberman "Monogamous prairie voles, which work frantically to reunite with separated mates, have far more oxytocin receptors in the nucleus accumbens…" 18:20 The neurochemical explanation sits cleanly in the oxytocin receptor data: monogamous voles have far more oxytocin receptors concentrated in the nucleus accumbens, the brain area governing motivation, craving, and pursuit. In these animals, attachment has been wired directly into the reward and drive system in a way that non-monogamous voles' brains have not. Huberman bridges this cleanly to human data: people who experience the most intense grief — the compulsive yearning, the reflexive impulse to reach out to the person — show the same oxytocin receptor pattern in brain imaging studies. The voles are not an analogy; they are a mechanistic preview of what happens in the human brain under grief.
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